This study is an extension of the UW Longitudinal Gene-Environment Study that focuses on ADHD specifically (i.e., 20 children with ADHD, 20 controls within the UW Longitudinal Gene-Environment Study) and the ways in which brain structure and functioning may help to explain the link between genes and ADHD. Linking genome-wide signals with neural function is crucial for understanding the biological mechanisms of risk for ADHD and may lead to novel targets for intervention. This study integrates genomics, bioinformatics data, and neuroimaging techniques to precisely characterize the brain mechanisms underlying genome-wide risk for ADHD.
Ryan Herringa, M.D., Ph.D. (Psychiatry)
Qiongshi Lu, Ph.D. (Biostatistics & Medical Informatics)